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Polioencephalomalacia (PEM), also referred … Polioencephalomalacia (PEM), also referred to as cerebrocortical necrosis (CCN), is a neurological disease seen in ruminants that is caused by multiple factors, one of which is thiamine depletion in the body. Thiamine (vitamin B1) is a key chemical in glucose metabolism that, when deficient, is most threatening to neurological activity. In addition to altered thiamine status, an association with high sulfur intake has been observed as a potential cause of PEM. Other toxic or metabolic diseases (eg, acute lead poisoning, sodium toxicosis/water deprivation) can result in PEM as well. Cattle, sheep, goat, and other ruminants that are diagnosed with PEM or pre-PEM suffer opisthotonus, cortical blindness, disoriented movement, and eventually fatality, if left untreated. Current data shows that the onset of PEM can range from birth to late adulthood.EM can range from birth to late adulthood.
, Die Zerebrokortikalnekrose (Cerebrocortica … Die Zerebrokortikalnekrose (Cerebrocorticalnekrose, CCN, „Hirnrindennekrose“) ist eine bei Wiederkäuern auftretende metabolisch-toxische Gehirnerkrankung (Enzephalopathie) infolge eines Mangels an Thiamin (Vitamin B1). Sie entspricht damit der Beriberi bzw. der Wernicke-Enzephalopathie des Menschen, der Chastek-Paralyse der Pelztiere und der Thiaminmangel-Enzephalopathie der Katze.r Thiaminmangel-Enzephalopathie der Katze.
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Die Zerebrokortikalnekrose (Cerebrocortica … Die Zerebrokortikalnekrose (Cerebrocorticalnekrose, CCN, „Hirnrindennekrose“) ist eine bei Wiederkäuern auftretende metabolisch-toxische Gehirnerkrankung (Enzephalopathie) infolge eines Mangels an Thiamin (Vitamin B1). Sie entspricht damit der Beriberi bzw. der Wernicke-Enzephalopathie des Menschen, der Chastek-Paralyse der Pelztiere und der Thiaminmangel-Enzephalopathie der Katze.r Thiaminmangel-Enzephalopathie der Katze.
, Polioencephalomalacia (PEM), also referred … Polioencephalomalacia (PEM), also referred to as cerebrocortical necrosis (CCN), is a neurological disease seen in ruminants that is caused by multiple factors, one of which is thiamine depletion in the body. Thiamine (vitamin B1) is a key chemical in glucose metabolism that, when deficient, is most threatening to neurological activity. In addition to altered thiamine status, an association with high sulfur intake has been observed as a potential cause of PEM. Other toxic or metabolic diseases (eg, acute lead poisoning, sodium toxicosis/water deprivation) can result in PEM as well. Cattle, sheep, goat, and other ruminants that are diagnosed with PEM or pre-PEM suffer opisthotonus, cortical blindness, disoriented movement, and eventually fatality, if left untreated. Current data shows that f left untreated. Current data shows that
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Zerebrokortikalnekrose
, Polioencephalomalacia
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